Even after six months without exposure, glyphosate’s harmful effects persist in brain tissue
Groundbreaking research reveals that exposure to glyphosate, America’s most widely used herbicide, leads to lasting brain inflammation and accelerates Alzheimer’s-like changes, even after a lengthy recovery period.
The active ingredient in common weed killers poses a significant threat to brain health, with new evidence showing its toxic effects persist long after exposure ends. Glyphosate’s breakdown product remains in brain tissue for at least six months, triggering a cascade of damaging effects on brain function and structure.
The herbicide’s impact proves particularly devastating for those at risk of Alzheimer’s disease. Research demonstrates it accelerates the buildup of harmful beta-amyloid plaques and tau proteins – hallmark features of Alzheimer’s. Beyond these structural changes, exposed subjects showed reduced survival rates and increased anxiety-like behaviors, indicating broader effects on brain function.
Most alarming, the chemical triggers persistent brain inflammation in both healthy and Alzheimer’s-prone subjects. With 83.87% of Americans already showing detectable levels in their urine and approximately 300 million pounds used annually in U.S. agriculture, these findings raise urgent concerns about public health. The evidence suggests that glyphosate exposure may be a previously unrecognized environmental risk factor for accelerated brain aging and neurodegeneration.
Source: Bartholomew SK, Winslow W, Sharma R, et al. Glyphosate exposure exacerbates neuroinflammation and Alzheimer’s disease-like pathology despite a 6-month recovery period in mice. J Neuroinflammation. 2024;21:316.
